Mina Kodsi

neurofibromas, schwannomas, gliomas, and pheochromocytomas (nf-1 = tumor suppressor)

mass crosses corpus callosum + high csf protein

cerebellopontine angle (good prognosis with resection)

t. pallidum/neurosyphilis (tabes dorsalis of dorsal column)

lewy bodies

head of caudate atrophy

neurofibrillary tangles + apo e4 = chrom 19, sorl = 11

perivascular lymphocytes + demyelination

fall and hemorrhage in gyrus rectus

benign (vertebrae don't close over cord)

lemon sign/banana sign (folate deficit)

occiput doesn't form = brain herniation (increased afp)

obstruct csf flow = hydrocephalus

linear parasagittal infarct

hemorrhage into basal ganglia

berry aneurysm --> subarachnoid hemorrhage

temporal lobe lesion

thiamine deficiency (periaqueductal gray)

in pons/midbrain from herniation --> edema + uncal herniation damaging small perforating vessels

cerebellar tonsil herniation through posterior fossa

biphasic synchronous sharp-wave complexes

meningomyelocele, cerebellar tonsil displaced down, medulla displaced from posterior fossa into foramen magnum, midbrain tectum is tenting

enlarged posterior cranial fossa with absent cerebellar vermis replaced by midline cyst

lose anterior horn cells (like als) --> "floppy baby"

taenia solium/pork tapeworm) = form cysts within subarachnoid space and cortex (non-enhanching lesions) --> can block csf = hydrocephalus

ring-enhancing lesion (most common in hiv)  

b. burgdorferi causes erythema migrans, pericarditis, and neuro symptoms

go to grey-white junctions

inside ventricles (well circumscribed) --> can cause obstructive hydrocephalus

sodium imbalance (siadh complication)